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New Alzheimer’s rat model will boost research
May 2013
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BETHESDA, Md.—Researchers funded by the U.S. National Institutes of Health have found that a new genetically engineered lab rat with the full complement of brain changes associated with Alzheimer's disease supports the hypothesis that beya-amyloid increase in the brain causes the disease. The rats used were engineered with mutant amyloid precursor protein and presenilin 1 genes, both of which are linked with early-onset Alzheimer's. Experiments confirmed neurofibrillary tangles in Alzheimer's-targeted brain regions and neuron death in those regions.  
 
"To fully benefit from this exciting new work, there is a critical need to share the animal model with researchers dedicated to finding ways to delay, prevent or treat Alzheimer's disease," said Dr. Neil Buckholtz of the National Institute on Aging. "Accordingly, Dr. Town and his colleagues are working towards making their new rat model easily accessible to the research community."

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